Baik, J. H. (2013). Dopamine signaling in Food Addiction: role of dopamine D2 receptors. BMB Rep, 46(11), 519-526. https://doi.org/10.5483/bmbrep.2013.46.11.207 Review Study link
The neurobiological underpinnings of obesity and binge eating: A rationale for adopting the Food Addiction model
Smith DG, Robbins TW. The neurobiological underpinnings of obesity and binge eating: A rationale for adopting the food addiction model. Biol Psychiatry 2013;73(9): 804-10. Abstract: The Food Addiction model of overeating has been proposed to help explain the widespread advancement of obesity over the last 30 years. Parallels in neural substrates and neurochemistry, as well as corresponding motivational and behavioral traits, are increasingly coming to light; however, there are still key differences between the two disorders that must be acknowledged. We critically examine these common and divergent characteristics using the theoretical framework of prominent drug addiction models, investigating the neurobiological underpinnings of both behaviors in an attempt to justify whether classification of obesity and binge eating as an addictive disorder is merited. Study link
If Sugar is Addictive, What Does it Mean for the Law?
Gearhardt, A., Roberts, M., Ashe, M. (2013). If Sugar is Addictive, What Does it Mean for the Law? Journal of Law, Medicine and Ethics 41(S1) 46-49, https://doi.org/10.1111/jlme.12038 “… the objective here is to propose an analytical framework that recognizes the role of politics and regulatory philosophy in evaluating the legal tools that could be employed if sugar-addiction research gains traction. This limited analysis seeks to raise core issues and stimulate a more robust and thorough discussion. gearhardt2013sugarlaw .pdf
The addictive dimensionality of obesity
Volkow, N. D., Wang, G. J., Tomasi, D., & Baler, R. D. (2013). The addictive dimensionality of obesity. Biological psychiatry, 73(9), 811—818. htt s: doi.or 10.1016 •.bio s ch.2012.12.020 Review Addictive_Dimensionality_of_Obesity .pdf
Food Addiction detox and abstinence reinterpreted?
Shriner, R. L. (2013). Food Addiction: detox and abstinence reinterpreted? Exp Gerontol, 48(10), 1068-1074. https://doi.org/10.1016/i.exger.2012.12.005 Opinion on metabolic inflammation shriner2013 .pdf
The drive to eat comparisons and distinctions between mechanisms of food reward and drug addiction
DiLeone, R., Taylor, J. & Picciotto, M. The drive to eat: comparisons and distinctions between mechanisms of food reward and drug addiction. Nat Neurosci 15, 1330–1335 (2012). https://doi.org/10.1038/nn.3202 Commonalities and differences in systems-level and behavioral responses to FA and drugs dileone2012 .pdf
Obesity and addiction: neurobiological overlaps
Volkow, N. D., Wang, G. J., Tomasi, D., & Baler, R. D. (2013). Obesity and addiction: neurobiological overlaps. Obesity reviews : an official journal of the International Association for the Study of Obesity, 14(1), 2-18. https://doi.org/10.1111/j.1467-789X.2012.01031.x Review volkow2012 .pdf
Rationale and consequences of reclassifying obesity as an addictive disorder: neurobiology, food environment and social policy perspectives
Allen, P. J., Batra, P., Geiger, B. M., Wommack, T., Gilhooly, C., & Pothos, E. N. (2012). Rationale and consequences of reclassifying obesity as an addictive disorder: neurobiology, food environment and social policy perspectives. Physiology & behavior, 107(1), 126—137. https://doi.org/10.1016/i.physbeh.2012.05.005 Review allen2012 .pdf
Dual roles of dopamine in food and drug seeking: the drive-reward paradox
Wise R. A. (2013). Dual roles of dopamine in food and drug seeking: the drive-reward paradox. Biological psychiatry, 73(9), 819—826. https://doi.org/10.1016/i.biopsych.2012.09.001 Review wise2013 .pdf
Obesity and the brain how convincing is the addiction model?
Ziauddeen, H., Farooqi, I. S., & Fletcher, P. C. (2012). Obesity and the brain: how convincing is the addiction model? Nature Reviews Neuroscience, 13(4), 279–286. doi:10.1038/nrn3212 An increasingly influential perspective conceptualizes both obesity and overeating as a food addiction accompanied by corresponding brain changes. Because there are far-reaching implications for clinical practice and social policy if it becomes widely accepted, a critical evaluation of this model is important. Weexamine the current evidence for the link between addiction and obesity, identifying several fundamental shortcomings in the model, as well as weaknesses and inconsistencies in the empirical support for it from human neuroscientific research. At the outset, it is important to acknowledge that the food-addiction literature has largely adopted the clinical model of addiction as defined by the DSM-IV. Although this model has clinical validity, in the addiction research literature it has been supplemented, and to an extent superseded, by powerful neurobiological models that have decomposed the clinical syndrome in terms of its core cognitive processes and their possible neural substrates (BOX 1). This approach, which is based on a growingunderstanding of the neurobiology of addiction, is welcome and — as we discuss — may offer new ways of identifying overlap between obesity and addiction. However, this article is primarily concerned with theexisting arguments in favour of addiction as a model for obesity, arguments that draw on clinical definitions. ziauddeen2012 .pdf